Relationship Between Insomnia And Depression Psychology Essay

Relationship Between Insomnia And Depression Psychology EssayNowadays, insomnia is the most widespread sleeping disorder of this century. Being 1 of the most common sleep disorders, insomnia rump be defined as inability to maintain a good sleep hygiene. According to epidemiological studies, atleast hotshot third of the general population suffer from sleep troubles and among this, the incidence rate of insomnia is around 6% to 10% (Daley et al. 2009). Insomnia piece of tail be viewed as either world a symptom or a syndrome the term secondary effect or symptom can be applied in cases set shoot by psychiatric afflictions, diseases, intake of illicit drugs or excess of alcohol and level off an aggregate of all these calculates coupled with stress, anxiety or stamp (Sivertsen et al. 2009). Insomnia can be evaluated using the Insomnia severity index (ISI) which establish on the patients providing an assessment of the intensity of their symptoms (Bastien et al, 2001). The Pittsburg h Sleep Quality Index (PSQI) which makes use of questionnaires is a practical manner to assess the sleep quality along with the troubles causing it over a certain period of time (Backhaus 2002). Insomnia is regularly linked to psychiatric disturbances such as depression. almost researches manoeuver that depression is the factor that causes insomnia while others disagree by stating that in fact, lidlesss atomic number 18 to a greater extent pr unmatched to develop depression (Isaac Greenwood 2011). Depression can be investigated using the Beck Depression Inventory (BDI). This literature review will be focused on defining the diametrical types of insomnia and whether or not they are caused by depression. The flow of the bidirectional relationship between insomnia and depression will also be presented to determine which one of these two is a find factor.Insomnia caused by depressionTypes of insomniaPrimary InsomniaPrimary insomnia (PI) is the repetitive inability to commence or uphold sleep and this excludes every natural or psychotic disturbances (Backhaus 2002). This has an impact on the quality of life of the patient causing distress and unfitness both socially and physically. Woods et al. 2008 reports that PI occurs in atleast 3% of the population in the westbound developed nations. According to a study conducted in the general population by Morin et al. (2006), muckle complaining of dissatisfaction in their sleep are more ilk to be developing insomnia symptoms compared to those pull in an appeasing and pleasant sleep. Some of the symptoms that might qualify a person as world insomniacs are difficulty to take asleep, boast trouble in upholding sleep, waking up early in the morning or have a non-regenerative sleep these symptoms can either issue individually or in combination with one another as shown in studies by Leger et al. (2010). PI does not occur due to any other particular health conditions. It is independent of other factors.Secondary InsomniaAs defined by the DSM (Diagnostic and Statistical Manual of Mental Disorders), secondary insomnia can be linked to mental disturbances, trouble to sleep owing to a medical state or stimulation by a substance. Secondary insomnia is one which is related to to another mental disturbance whereby one of the chief grievances is based on the inability of start or maintains a proper sleep and that the sleep even of it is initiated is not rejuvenating at all and this spans for over a period of atleast one month. It occurs in close collaboration with other psychiatric and medical conditions whereby clinical depression is one of the main concern, this state is considered as a causative agent for insomnia.Transient or Acute InsomniaInsomnia can be classifies as being short-lived or acute is the delay of sleep disturbance expand over a period of less than a few nights or not more than three to four weeks (Fetveit et al. 2008). This type of insomnia is more apt(predicate) to occur in p eople who have no previous history of sleep disorders but who have been exposed to some distinctive cause such as caffeine intake, nicotine or any other medications.Chronic InsomniaChronic insomnia can be construe in two different ways either as a syndrome as a whole similar to PI or as being the consequence arising from a medical ailment such as secondary insomnia or mental imbalance such as major depression, anxiety or by overdose of drugs and alcohol (de Sainte Hilaire et al. 2005). In relation to Fetveit et al. (2008), primary insomnia caters for around 25% of all the cases of chronic insomnia.2.2 The Mechanism of how Depression causes InsomniaDepression is a factor that triggers insomnia by causing imbalance or deficiency in terms of serotonin which is monoamine neurotransmitter. These particular serotonergic activies govern the sleep regulation. According to studies conducted in vivo by Joensuu et al. (2007), the availability of the serotonin transporter (SERT) changes at di fferent stages of depression and this can be turn up by using a technique known as SPECT (Single proton electric arc tomography). This decline in terms of SERT which is due to depression is mostly localised in the diencephalon in the mid part of the brain (Joensuu et al. 2007). This decline in terms of serotonin level negatively influences the sleep patterns since the serotonin is key constituent affecting both the sleep phases and mood (Buckley Schatzberg 2010). The hypersecretion of cortisol along with adrenocorticotropic hormone is an indicator of insomnia. Changes made to the Hypothalamic-Pituitary-Adrenal (HPA) axis along with the mode of action of the hormone that predicts the start of sleep triggers the onset of depression. The hormone melatonin keeps the circadian rhythm of the body constant. Some studies show that depression causes the level of melatonin to drop which in turn causes a domino effect on the serotonin level. In people with major depressive syndromes, the denseness of cortisol is much higher than that of melatonin (Buckley Schatzberg 2010) as shown in the graphs below which highlights the relationship between the level cortisol and melatonin.Fig. 2 shows the phase relationship between cortisol and melatonin for healthy control compared to the one miserable from mental disorder (Buckley Schatzberg 2010).Yet, there are still other numerous neurobiological mechanisms like the deficiency of the monoamine neurotransmitters, undue activity in the HPA axis, the faulty action of the gene related to plasticity and circadian genes mutation that can give an explanation on the irregularity in terms of sleep patterns in depressive patients (Benca Peterson, 2008). This diagnosis of sleeping troubles using only polysomnography is not reliable and must be supported by functional imaginativeness or EEG activity patterns to define the correlation between the behavioral and experimental observations (Benca Peterson 2008).The bidirectional flow of the relationship between insomnia and depressionDepression causes insomniaIn those people who suffer from MDD, one of their major grievances is insomnia. In the adult population, around 60% of them that fits the criteria defining MDD whine about insomnia and an average of 10% to 20% of the insomniacs show signs of MDD (Fava et al. 2006). Using multiple variances, the mean values mean response for two particular groups (insomniacs with or without symptoms of depression) responses pertaining t to BDI-II were analyzed. From the total score derived from the BDI-II, it was seen that depressed plus insomniac participants attained much higher grades on the total score scale in contrast to those with solely insomnia. A feature that highlights depression is the shift in the sleep pattern that leading to insomnia and other disturbances. Among 90% of the depressive patients are insomniacs as well according to a study by Fava et al. (2006), this clearly emphasize on the co-existence of depress ion and insomnia.Insomnia triggers depressionThough not the only one, insomnia is regarded as being the main factor that has an impact on depression. Studies conducted by Pigeon et al. (2008), patients with insomnia are more likely to become depressed and remain so. From the cohort study obtained from health surveys of the HUNT-2 by Neckelmann et al. (2007), the relationship between insomnia and depression is simply based on their resemblance to one another. This defines insomnia as a state marker for depression. As established by Taylor et al. (2005) with BDI and sleep diaries to support the research, the probability of insomniacs developing depression was 9.82 times more than people without insomnia as presented by the table 1. Further, this same study states that the depression score for patients with combined insomnia is much higher compared to other studies.Table 1. Prevalence Rates evince using the Beck Depression Inventory Score to relate insomnia and depression (Taylor et a l. 2005).From the direct analysis of figure 3 below that 4 out of the 5 different depression statuses prevail from persistent insomnia while more than 50% of the sample did not remit or have an improvement from the condition (Pigeon et al. 2008). This proves that insomnia (persistent or intermediate) does have a significant impact on the number of people with improved condition. All these facts boil down to the fact that insomnia is certainly a major risk factor for depression.Fig. 3 highlights the relationship of insomnia and how it is a risk factor for depression (Pigeon et al. 2008)DiscussionDifferent types of insomnia react differently when exposed to other factors such as MDD for example it does not in any way cause primary insomnia however in cases of secondary insomnia, it is considered as being the most crucial factors based on studies related to DSM.This study did not really determine whether a correlation exists between insomnia and depression since they have common sympto ms, yet the use of BD1-II did observe the symptoms of depression in insomniacs.The PSQI has a high sensitivity and specificity forinsomnia patients in comparison to healthy controls, thusunderscoring that it is a good measure for differentiatingbetween good sleepers and patients suffering from sleepdisturbances. Our data suggest, however, that the cut-offscore should be set to 6 in order to maximize specificitywhile only modestly reducing sensitivity. In sum, the PSQIproved to be a valuable adjunct to clinical work on insomniaand is a useful first-line, easy-to-handle, and time-efficientquestionnaire to evaluate sleep disturbances.Conclusion